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If he does have it, he very well may not know it, since many men with the syndrome go through life undiagnosed. Plus, if he were to be diagnosed by a physician, he could get treatment in the form of testosterone therapy, which could help boost his self-confidence and improve his physical appearance. They have been dating for a while now, and apparently he has never mentioned anything about it. Could you maybe have this disorder I read about on the internet? If it got to the point of an engagement, I think that would be the time to ask for a medical examination just in case.
If I was that woman, I would simply break up with him if it would bother me that much. Marriage is for better or for worse. I would think you should be very close to that person before ever mentioning this.
13 year old Braydon With Klinefelter syndrome
Then it should be done privately, and with the strong assurance that you mean nothing cruel or unkind by asking. Otherwise, I would not mention this.
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Yup, and I still believe that if God is at the center of the relationship, His grace will make it work. If this woman suspects he has it and does not want to marry him because of that, she should just walk away rather then get him tested and then say goodbye if the test is positive.
And if the test is negative, and she stays with him, this guy would never be able to fully trust her. Maybe she just wants a valid, consummated marriage, to have a husband she can enjoy the normal joys of married life with and not to have the embarrassment and inconvenience of an annulment? After all, the only way a Catholic ever should find out if they can consummate a marriage is to wait until the wedding night.
If that ever happened to me, I would consider it fraud on the marriage and seek to have it annulled on that basis. Note I did not say I want to attempt the consummation before the wedding, no I want to make sure that she is on board with consummation happening that night, and if she has a condition that could interfere, I need to know before the engagement.
If that results in me breaking up with her, better that than her breaking my heart after the wedding with her coverup. I would encourage your future wife to be upfront and tell you about all the issues she has before the marriage because as you eluded into, it would be dishonest not to.
However, after reading about the syndrome, I have to wonder what on earth would make this woman suspect her boyfriend had it because there are no signs.
I Klinefelter Dating have been taken home by a woman 15 years older than me from a pub. I Klinefelter Dating have had orgies with friends I Klinefelter Dating have known for over a / Aug 05, If you are dating a man you suspect has Klinefelter's syndrome, is there a sensitive, respectful way to broach the subject without embarrassing him or making him feel . The first steps back in the world of dating should not to be taken lightly. You shouldn't attempt to bed new women to take revenge on your ex, whether it's to lure her back with jealousy or as /
A wise person ensures they can trust their future spouse. I see two scenarios here. Unless the OP explains where this concern is coming from, I have to really wonder why anyone would suspect that of a man.
I am giving the man the benefit of the doubt. Mayo Clinic does not endorse companies or products.
Advertising revenue supports our not-for-profit mission. This content does not have an English version. This content does not have an Arabic version. Overview Klinefelter syndrome is a genetic condition that results when a boy is born with an extra copy of the X chromosome.
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Klinefelter syndrome. Mayo Clinic; Genetics Home Reference. Accessed Aug. About Klinefelter syndrome. National Human Genome Research Institute.
Klinefelter syndrome 47,XXY. Merck Manual Professional Version. Klinefelter syndrome KS. Genetic and Rare Diseases Information Center.
Bearelly P, et al. By adulthood, XXY males look similar to males without the condition, although they are often taller. In adults, possible characteristics vary widely and include little to no sign of affectedness, a lankyyouthful build and facial appearance, or a rounded body type with some degree of gynecomastia increased breast tissue.
Affected males are often infertileor may have reduced fertility. Advanced reproductive assistance is sometimes possible.
Because of this primary hypogonadism, individuals often have a low serum testosterone level, but high serum follicle-stimulating hormone and luteinizing hormone levels. XXY males are also more likely than other men to have certain health problems, such as autoimmune disordersbreast cancervenous thromboembolic diseaseand osteoporosis.
Some degree of language learning or reading impairment may be present,  and neuropsychological testing often reveals deficits in executive functionsalthough these deficits can often be overcome through early intervention. Maternal age is the only known risk factor. The extra chromosome is retained because of a nondisjunction event during paternal meiosis Imaternal meiosis Ior maternal meiosis II gametogenesis. The relevant nondisjunction in meiosis I occurs when homologous chromosomes, in this case the X and Y or two X sex chromosomes, fail to separate, producing a sperm with an X and a Y chromosome or an egg with two X chromosomes.
Another mechanism for retaining the extra chromosome is through a nondisjunction event during meiosis II in the egg. Nondisjunction occurs when sister chromatids on the sex chromosome, in this case an X and an X, fail to separate. This XXY chromosome arrangement is one of the most common genetic variations from the XY karyotype, occurring in about one in live male births.
In mammals with more than one X chromosome, the genes on all but one X chromosome are not expressed; this is known as X inactivation.
Additional chromosomal material can contribute to cardiac, neurological, orthopedic, and other anomalies. Thus far, only about 10 cases have been described in literature. Analogous XXY syndromes are known to occur in cats -specifically, the presence of calico or tortoiseshell markings in male cats is an indicator of the relevant abnormal karyotype. As such, male cats with calico or tortoiseshell markings are a model organism for KS, because a color gene involved in cat tabby coloration is on the X chromosome.
The standard diagnostic method is the analysis of the chromosomes' karyotype on lymphocytes. A small blood sample is sufficient as test material.
In the past, the observation of the Barr body was common practice, as well. Physical characteristics of a Klinefelter syndrome can be tall stature, low body hair and occasionally an enlargement of the breast. There is usually a small testicle volume of ml per testicle standard values: ml. A spermiogram can also be part of the further investigation. Often there is an azoospermia present, rarely an oligospermia. The genetic variation is irreversible, thus there is no causal therapy.
From the onset of puberty, the existing testosterone deficiency can be compensated by appropriate hormone replacement therapy. If gynecomastia is present, the surgical removal of the breast may be considered for both the psychological reasons and to reduce the risk of breast cancer.
The use of behavioral therapy can mitigate any language disorders, difficulties at school, and socialization. An approach by occupational therapy is useful in children, especially those who have dyspraxia. Methods of reproductive medicine, such as intracytoplasmic sperm injection ICSI with previously conducted testicular sperm extraction TESEhave led to men with Klinefelter's syndrome to produce biological offspring.
The lifespan of individuals with Klinefelter syndrome appears to be reduced by approximately 2. This syndrome, evenly distributed in all ethnic groupshas a prevalence of one to two subjects per every males in the general population.
The syndrome is also the main cause of male hypogonadism. The syndrome was named after American endocrinologist Harry Klinefelterwho in worked with Fuller Albright and E. Reifenstein at Massachusetts General Hospital in Boston, Massachusettsand first described it in the same year. Considering the names of all three researchers, it is sometimes also called Klinefelter-Reifenstein-Albright syndrome.
Jacobs described her discovery of this first reported human or mammalian chromosome aneuploidy in her William Allan Memorial Award address. Klinefelter syndrome can also occur in other animals.
In cats it can result in a male tortoiseshell and calico cat patches of different colored fura pattern that is usually only seen in female cats.
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